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SNU-398人肝癌細胞(附S

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人肝癌細胞SNU-398種屬人別稱SNU398; NCI- SNU- 39 8組織來源肝臟疾病肝細胞癌傳代比例/細胞消化1:2傳代 ,消化1-3分鐘,培養基配置RPMI1640 培養基: 10%胎牛血清

人肝癌細胞SNU-398

種屬
別稱SNU398; NCI- SNU- 39 8
組織來源肝臟
疾病肝細胞癌
傳代比例/細胞消化1:2傳代 ,消化1-3分鐘 ,
培養基配置RPMI1640 培養基: 10%胎牛血清 1%雙抗
簡介SNU-398 于 1990 年由 J.-G. Park 及其同事取自一名韓國患者的間變性肝細胞癌 ,該患者已通過脂質體加 和-C 的組合進行了經導管動脈栓塞治療。既可以附著細胞也可以懸浮細胞。懸浮的細胞是可行 ,不應丟 棄。通過溫和離心 (125 xg) 回收懸浮細胞 ,以與貼壁細胞群一起進行繼代培養。
形態上皮細胞樣
生長特征懸浮貼壁生長
倍增時間每周 2 3
抗原表達Blood Type O; Rh +
STRAmelogenin: X,Y CSF1PO: 13 D13S317: 11 D16S539: 10, 14 D5S818: 12 D7S820: 10, 11 THO1: 7,9 TPOX:11 vWA: 17, 18
保藏機構ATCC; CRL- 2233

Background: Hospitals face mounting pressure to reduce unplanned utilization amid rising healthcare demands from an aging population. The Case management for At-Risk patients in the Emergency Department (CARED) program is among the first ED transitional care strategies to focus on both frail older adults and Emergency Department (ED) re-attenders to reduce acute hospital utilization. This study aims to evaluate the effectiveness of the CARED program in reducing hospital (re)admissions and ED re-attendances within 30- and 60-days post-discharge.

Methods: A retrospective, propensity-matched study was conducted from April 2022 to July 2023 in the ED of Ng Teng Fong General Hospital in Singapore. The CARED program identifies and enrols at-risk patients i.e., frail older adults and patients who re-attend the ED within 30 days of hospital discharge, for a geriatric assessment. This is followed by multidisciplinary team care, discharge planning and right siting of care from the ED to community-based services by ED case managers. The primary outcomes were hospital (re)admissions and ED re-attendances within 30- and 60-days post-discharge. Secondary outcomes were cost avoidance and bed occupancy days from reduced acute hospital usage.



untreated host, most aeTSAs are undetected by the immune system. We present evidence suggesting that vaccines inducing direct aeTSA presentation by DCs may represent an attractive strategy for cancer treatment.Checkpoint inhibitors (eg, programmed cell death protein 1 [PD-1], programmed cell death ligand 1 [PD-L1], cytotoxic T-lymphocyte associated protein 4 [CTLA-4] antibodies) are changing how we understand cancer and provide a means to develop modern

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